|
|
|
| Analysis of the Causes of Short Stature in School-age Children in Panyu District, Guangzhou |
| WENG Dian-zi |
| Panyu District He Xian Memorial Hospital,, Guangzhou 511400, China |
|
|
|
|
Abstract 【Objective】To evaluate the detailed etiology of school-age children with short stature in Panyu District, Guangzhou. 【Methods】The clinical data of 150 school-age (6-14-year-old) children with short stature were collected by mobile phone from July 1, 2018 to July 1, 2020 in the Pediatric Endocrinology Department of He Xian Memorial Hospital, Panyu District, Guangzhou city. The etiological factors of school-age children with short stature in this area were analyzed retrospectively, and the etiological distribution of children with different gender and age were analyzed retrospectively. 【Results】In Panyu District of Guangzhou, idiopathic dwarfism accounted for the highest proportion (55.33%), followed by growth hormone deficiency (30.67%), followed by familial dwarfism (4.00%), pituitary tumor (1.33%), hypophyseal dysplasia (3.33%), achondroplasia (0.67%), Turner syndrome (1.33%), small for gestational age (2.67%), and chronic renal insufficiency Tubular acidosis (0.67%). In children with short stature, male children were more common (69.33%), girls were less common (30.67%), idiopathic short stature and growth hormone deficiency were more common in male children (36.67% and 24.67%, respectively). Children aged 8-12 accounted for 59.33%, followed by children aged 6-8 and 12-14 (23.33% and 17.33%, respectively). Idiopathic short stature and growth hormone deficiency were mainly 8-12-year-old children (32.67% and 22.67%, respectively), while children aged 6-8 and 12-14 years accounted for 16.00%, 5.33% and 6.67% and 4.67% respectively.【Conclusion】There are many causes of school-age short stature in Panyu District of Guangzhou city. Idiopathic short stature and growth hormone deficiency are the main causes. Familial dwarfism, hypophysis hypoplasia and small for gestational age infants are also included.
|
|
Received: 29 June 2020
|
|
|
|
|
|
[1] 韩晓伟,董治亚,张婉玉,等.矮小症病因及临床特征分析[J].临床儿科杂志,2019,37(1):39-42.
[2] 刘朔.NPR2基因与矮小症关系的研究进展[J].天津医科大学学报,2020,26(1):86-90.
[3] 王继锋,张薇,马勇,等.儿童矮身材病因及其骨龄发育情况分析[J].中国妇幼保健,2019,34(6):1288-1290.
[4] 王卫平. 儿科学[M]. 北京:高等教育出版社, 2004:327-331.
[5] 李辉.中国0～18岁儿童、青少年身高、体重的标准化生长曲线[J].中华儿科杂志,2009,47(7):487-492.
[6] 吴韬,杜俊文,张坤,等.儿童矮小症485例病因探讨及治疗效果[J].河北医科大学学报,2018,39(12):1384-1388.
[7] 张磊,王耀邦,高凤,等.生长激素-胰岛素样生长因子轴功能检测在矮小症儿童病因诊断中的意义[J].临床和实验医学杂志,2019,18(22):2441-2444.
[8] 王贵莲,胡冰,张招文,等.宜春市矮小症儿童骨龄发育情况及病因分析[J].当代医学,2019,25(19):151-153.
[9] 潘晓琴,谭传梅,唐红艳,等.常德地区儿童矮小症136例病因分析[J].医学理论与实践,2019,32(20):3366-3367.
[10] 徐超.生长激素缺乏症矮小儿童垂体的MRI表现及常见病因分析[J].实用放射学杂志,2019,35(4):621-625.
[11] 解欣,苏延,王玉,等.Ghrelin、Nesfatin-1与儿童矮小症的相关性研究[J].医学理论与实践,2019,32(19):3048-3050. |
|
|
|
2020, Vol. 37 
