[1] 王福顺. 加强对创伤性脑损伤后早期癫痫机理的研究[J].中华神经外科疾病研究杂志, 2016, 15(4):289-292.
[2] Wang KY, Yu GF, Zhang ZY, et al . Plasma high-mobility group box 1 levels and prediction of outcome in patients with traumatic brain injury[J]. Clin Chim Acta(Engl) , 2012, 413(21-22):1737.
[3] 夏冬剑, 刘兴波, 张状. 硫酸镁对创伤性脑损伤大鼠兴奋性氨基酸和脑水肿的影响[J].中国新药杂志, 2010,19(23):2181-2184.
[4] Balu R.Inflammation and immune system activation after traumatic brain injury[J]. Curr Neurol Neurosci Rep, 2014, 14(10):484.
[5] Garred P, Honoré C, Ma YJ, et al . MBL2, FCN1, FCN2 and FCN3-The genes behind the initiation of the lectin pathway of complement[J]. Mol Immunol, 2009, 46(14):2737-2744.
[6] Brennan FH.Complement activation in the injured central nervous system: another dual-edged sword[J] ? J Neuroinflammation, 2012, 9(1):137.
[7] Hein E, Honorã C, Skjoedt MO, et al . Functional analysis of Ficolin-3 mediated complement activation[J]. PLoS One, 2012, 5(11):e15443.
[8] Füst G, Munthe-Fog L, Illes Z, et al . Low ficolin-3 levels in early follow-up serum samples are associated with the severity and unfavorable outcome of acute ischemic stroke[J]. J Neuroinflammation, 2011, 8(1):1-10.
[9] Yanamadala V, Friedlander RM.Complement in neuroprotection and neurodegeneration[J]. Trends Mol Med, 2010, 16(2):69-76.
[10] Pan JW, Gao XW, Hao J, et al . Low serum ficolin-3 levels are associated with severity and poor outcome in traumatic brain injury[J]. J Neuroinflammation, 2015, 12(1):226-233.
[11] Zanier ER, Zangari R, Munthefog L, et al . Ficolin-3-mediated lectin complement pathway activation in patients with subarachnoid hemorrhage[J]. Neurology, 2014, 82(2):126-134.
[12] Munthe-Fog L, Hummelsh JT, Honor C, et al . Immunodeficiency associated with FCN3 mutation and Ficolin-3 deficiency[J]. N Engl J Med, 2009,360(25):2637-2644. |
2018, Vol. 35 
