[1] 王晓燕, 花宝金, 李卫东. 炎症性肠病发病机制及治疗的研究进展[J]. 国际消化病, 2017, 37(5): 278-281. [2] Rajabian Z.Over-expression of immunosuppressive molecules, PD-L1 and PD-L2, in ulcerative colitis patients[J].Iran J Immunol,2019,16(1): 62-70. [3] Kao SC, Cheng YY, Williams M,et al. Tumour suppressor microRNAs contribute to the regulation of PD-L1 expression in malignant pleural mesothelioma[J].J Thorac Oncol,2017, 7(6): 556-562. [4] Alex P, Zachos NC, Nguyen T, et al. Distinct cytokine patterns identified from multiplex profiles of murine DSS and TNBS-induced colitis[J].Inflamm Bowel Dis,2009, 15(3): 341-352. [5] Jackson LN, Zhou Y, Qiu S, et al. Alternative medicine products as a novel treatment strategy for inflammatory bowel disease[J].Am J Chin Med,2008, 36(5): 953-965. [6] Menachem Y, Trop S, Kolker O, et al. Adoptive transfer of NK 1.1+ lymphocytes in immune-mediated colitis: a pro-inflammatory or a tolerizing subgroup of cells[J].Microbes Infect,2005, 7(5-6): 825-835. [7] 冯显清, 梁川, 刘然, 等. 双歧杆菌三联活菌胶囊联合美沙拉嗪对溃疡性结肠炎患者免疫功能及肠黏膜屏障功能的影响[J].国际消化病, 2018, 38(2): 144-147. [8] Gianchecchi E, Delfino DV, Fierabracci A. Recent insights into the role of the PD-1/PD-L1 pathway in immunological tolerance and autoimmunity[J].Autoimmun Rev,2013, 12(11): 1091-1100. [9] Wang X, Wu M, Cao Y, et al. Exploring the role of programmed cell death protein 1 and its ligand 1 in eye diseases[J].Crit Rev Clin Lab Sci,2019, 5(4): 1-15. [10] Fu Q, Man X, Wang X, et al. CD83+ CCR7+ NK cells induced by interleukin 18 by dendritic cells promote experimental autoimmune uveitis[J].J Cell Mol Med,2018, 6(7):56-67. [11] Tao Z, Xu S, Ruan H, et al. MiR-195/-16 Family Enhances Radiotherapy via T Cell Activation in the Tumor Microenvironment by Blocking the PD-L1 Immune Checkpoint[J].Cell Physiol Biochem,2018. 48(2): 801-814. [12] Jia X, Li X, Shen Y, et al. MiR-16 regulates mouse peritoneal macrophage polarization and affects T-cell activation[J].J Cell Mol Med,2016. 20(10): 1898-1907.
2019, Vol. 36 
