[1]Xu Y,Wang L,He J,et al.Prevalence and control of diabetes in Chinese adults[J].JAMA,2013,310(9):948-959. [2]Sharma AM,Wilks J,Aronow HD.Recurrent coronary artery thrombus formation in the setting of diabetic ketoacidosis[J].J Invasive Cardiol,2011 ,23(2):E1- E 4. [3]Cherian SV,Khara L,Das S,et al.Diabetic ketoacidosis complicated by generalized venous thrombosis: a case report and review[J].Blood Coagul Fibrin,2012,23 (3): 238. [4]苏敏,薛莉.脂蛋白磷脂酶A2 在2型糖尿病及其大血管病变中的作用[J].中国现代医药,2015,17(10):55-57. [5]殷艳.血浆脂蛋白相关磷脂酶A2 及纤维蛋白原与2 型糖尿病大血管病变的相关性分析[J].临床误诊误治,2016,29 (3):113-115. [6]Rosenson RS,Hurt-Camejo E.Phospholipase A2 enzymesand the risk of atherosclerosis [J].Eur Heart J,2012,33(23):2899-2909. [7]Katan M.Lipoprotein-associated phospholipase A2 is associated with atherosclerotic stroke risk: the Northern Manhattan Study[J].PLoS One,2014,9(1):e 83393. [8]王艺颖,张雪莹.2型糖尿病视网膜病变病人血栓前状态标志物检测[J].糖尿病新世界,2014,10(20):21. [9]郑伟成,肖卫民.缺血性卒中患者纤维蛋白原与动脉粥样硬化的相关性研究进展[J].中华脑血管病杂志,2011,5(4):316-321. [10]张良峰,孟庆利.血清总蛋白、白蛋白及纤维蛋白原水平与颈动脉粥样硬化程度的关系[J].中国老年学杂志,2014,34(16):4655-4656. [11]Corrado E,Rizzo M,Gppola G,et al.An update on the role of markes of inflammation in atheroxclerosis [J].Atheroscler Thromb,2010,17(1):1-11. [12]Chanavatian S,Stein RA,Atar D,et al. The course of D-dinner,high-sensitivity C-reactive protein and pro-B-type natriuretic peptide in patients with non-ST-elevation myocardial infarction [J].Clin Lab,2011,57(9-10):771-776. [13]Lowe G.Can haemostatic factors predict atherothronbosis[J].Intern Emerg Med,2011,6(6): 497-501. [14]王磊,章莹,费世早,等.动脉粥样硬化性脑梗死患者血浆脂蛋白相关磷脂酶A2 水平及相关性研究[J].卒中与神经疾病.2011,18(3):158-161.